纳豆激酶抑制DSS诱导的小鼠慢性结肠炎研究任务书

 2021-10-21 17:13:32

1. 毕业设计(论文)的内容和要求

1.封面:由学校统一印刷,按要求填写。

2.论文题目论文题目应该用简短、明确的文字写成,通过标题把毕业设计(论文)的内容、专业特点概括出来。

题目字数要适当,一般不宜超过20个字。

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2. 参考文献

[1] L.J. Dixon, A. Kabi, K.P. Nickerson, C. McDonald, Combinatorial effects of diet and genetics on inflammatory bowel disease pathogenesis, Inflamm. Bowel Dis. 21 (4) (2015) 912922.[2] R.J. Xavier, D.K. Podolsky, Unravelling the pathogenesis of inflammatory bowel disease, Nature 448 (7152) (2007) 427434.[3] Kakodkar S, Mutlu EA. Diet as a therapeutic option for adult inflammatory bowel disease[J]. Gastroenterol Clin North Am, 2017, 46 (4) : 745-767.[4] Sumi H, Hamada H, Tsushima H, et al.A novel fibrinolytic enzyme (nattokinase) in the vegetable cheese natto;a typical and popular soybean food in the Japanese diet[J].Experientia, 1987, 43 (10) :1110.[5] M. Fujita,Y. Ito,K. Hong,S. Nishimuro. Characterization of nattokinase-degraded products from human fibrinogen or cross-linked fibrin[J]. Fibrinolysis and Proteolysis,1995,9(3).[6] Chen H, Mcgowan E M, Ren N, et al.Nattokinase:a promising alternative in prevention and treatment of cardiovascular diseases[J].Biomarker Insights, 2018, 13:1-8.[7]Hsu R L, Lee K T, Wang J H, et al.Amyloid-degrading ability of nattokinase from Bacillus subtilis natto[J].J Agric Food Chem, 2008, 57:503-508.[8] Danese, S. Fiocchi, C. Ulcerative colitis. N. Engl. J. Med. 365,17131725 (2011).[9] Baumgart, D.C. s disease. Lancet 380,15901605 (2012).[10] Neurath, M.F. Cytokines in inflammatory bowel disease. Nat. Rev. Immunol.14, 329342 (2014).[11M.F. Neurath, Cytokines in inflammatory bowel disease, Nature 14 (2014)329342.][12] T. Hibi, N. Inoue, H. Ogata, M. Naganuma, Introduction and review: recent advances in the immunotherapy of inflammation bowel disease, J[13] T. Hayashi, T. Ishida, S. Motoya, F. Itoh, T. Takahashi, Y. Hinoda, K. Imai,Mucins and immune reactions to mucins in ulcerative colitis, Digestion 63(2001) 2831.[14] F.S. de Medina, I. Romero-Calvo, C. Mascaraque, O. Martinez-Augustin,Intestinal inflammation and mucosal barrier function, Inflamm. Bowel Dis. 20(2014) 23942404.[15] T. Nunes, C. Bernardazzi, H.S. de Souza, Cell death and inflammatory bowel diseases: apoptosis, necrosis, and autophagy in the intestinal epithelium,Biomed.Res. Int. 2 (2014) 218493.[16] S. Nikolaus, B. Schulte, N. Al-Massad, F. Thieme, D.M. Schulte, J. Bethge, et al.,Increased tryptophan metabolism is associated with activity of inflammatory bowel diseases, Gastroenterology 153 (6) (2017) 1504- .[17] N. Nam Trung, T. Nakahama, L. Duc Hoang, S. Le Van, C. Ha Hoang, T. Kishimoto, Aryl hydrocarbon receptor and kynurenine: recent advances in autoimmune disease research, Front. Immunol. 5 (2014).[18] Zelante T, Iannitti RG, Cunha C, et al. Tryptophan catabolites from microbiota engage aryl hydrocarbon receptor and balance mucosal reactivity via interleukin-22. Immunity 2013;39:372385.[19] Weng Y.Q., Yao J., Sparks S., et al.Nattokinase:an oral antithrombotic agent for the prevention of cardiovascular disease[J].International Journal of Molecular Sciences, 2017, 18 (3) :523.[20] The effects of nattokinase supplementation oncollagenepinephrine closure time, prothrombin time and activated partial thromboplastin time in nondiabetic and hypercholesterolemic subjects ,Food Funct., 2019, 10,2888[21] Kume K,Yamasaki M,Tashiro M,et al.Activations of coagulation andfibrinolysissecondary to bowel inflammation in patients with ulcerativecolitis[J].Intern Med,2007,46(17):1323-1329[22]牟小超 .纳豆激酶活性和稳定性的化学研究[D].太原.山西大学,2017.

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